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BPC-157 primarily modulates the nitric oxide system, promotes angiogenesis through VEGFR2 signaling, and protects mucosal surfaces
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Doses continue to escalate to 10mg and 12.5mg weekly
Taking all this data together it seems logical or appropriate to conclude that, because IGF1-R has a different signalling pathway that can maintain lipid oxidation in the liver, FFA uptake in muscle, and activates mTORC1 could reactivate IR through tyrosine kinase activity on IRS, thus displacing serine phosphorylation, reinstituting insulin signalling
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